ロード中...
β Adrenergic Receptor Kinase C-Terminal Peptide Gene-Therapy Improves β(2)-Adrenergic Receptor-Dependent Neoangiogenesis after Hindlimb Ischemia
After hindlimb ischemia (HI), increased catecholamine levels within the ischemic muscle can cause dysregulation of β(2)-adrenergic receptor (β(2)AR) signaling, leading to reduced revascularization. Indeed, in vivo β(2)AR overexpression via gene therapy enhances angiogenesis in a rat model of HI. G p...
保存先:
| 出版年: | J Pharmacol Exp Ther |
|---|---|
| 主要な著者: | , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
The American Society for Pharmacology and Experimental Therapeutics
2016
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6047230/ https://ncbi.nlm.nih.gov/pubmed/26604244 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1124/jpet.115.228411 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|