β Adrenergic Receptor Kinase C-Terminal Peptide Gene-Therapy Improves β(2)-Adrenergic Receptor-Dependent Neoangiogenesis after Hindlimb Ischemia

After hindlimb ischemia (HI), increased catecholamine levels within the ischemic muscle can cause dysregulation of β(2)-adrenergic receptor (β(2)AR) signaling, leading to reduced revascularization. Indeed, in vivo β(2)AR overexpression via gene therapy enhances angiogenesis in a rat model of HI. G p...

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Detalhes bibliográficos
Publicado no:J Pharmacol Exp Ther
Main Authors: Cannavo, Alessandro, Liccardo, Daniela, Lymperopoulos, Anastasios, Gambino, Giuseppina, D’Amico, Maria Loreta, Rengo, Franco, Koch, Walter J, Leosco, Dario, Ferrara, Nicola, Rengo, Giuseppe
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Pharmacology and Experimental Therapeutics 2016
Assuntos:
Cardiovascular
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6047230/
https://ncbi.nlm.nih.gov/pubmed/26604244
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1124/jpet.115.228411
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