Unfolded protein response (UPR) signaling regulates arsenic trioxide-mediated macrophage innate immune function disruption

Arsenic exposure is known to disrupt innate immune functions in humans and in experimental animals. In this study, we provide a mechanism by which arsenic trioxide (ATO) disrupts macrophage functions. ATO treatment of murine macrophage cells diminished internalization of FITC-labeled latex beads, im...

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Библиографические подробности
Опубликовано в: :Toxicol Appl Pharmacol
Главные авторы: Srivastava, Ritesh K., Li, Changzhao, Chaudhary, Sandeep C., Ballestas, Mary E., Elmets, Craig A., Robbins, David J., Matalon, Sadis, Deshane, Jessy S., Afaq, Farrukh, Bickers, David R., Athar, Mohammad
Формат: Artigo
Язык:Inglês
Опубликовано: 2013
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Article
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC6028020/
https://ncbi.nlm.nih.gov/pubmed/23954561
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.taap.2013.08.004
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