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Trpc6 inactivation confers protection in a model of severe nephrosis in rats
ABSTRACT: Mutations in canonical transient receptor potential-6 (TRPC6) channels give rise to rare familial forms of focal and segmental glomerulosclerosis (FSGS). Here we examined a possible role for TRPC6 in the progression of chronic puromycin aminonucleoside (PAN) nephrosis in Sprague-Dawley rat...
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| Publié dans: | J Mol Med (Berl) |
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| Auteurs principaux: | , , |
| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
Springer Berlin Heidelberg
2018
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6015123/ https://ncbi.nlm.nih.gov/pubmed/29785489 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00109-018-1648-3 |
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