Loss of Axon Bifurcation in Mesencephalic Trigeminal Neurons Impairs the Maximal Biting Force in Npr2-Deficient Mice

Ítems similars

• Bifurcation of Axons from Cranial Sensory Neurons Is Disabled in the Absence of Npr2-Induced cGMP Signaling
  per: Ter-Avetisyan, Gohar, et al.
  Publicat: (2014)
• Receptor guanylyl cyclase B-mediated cGMP signalling controls axon bifurcation of cranial sensory ganglion neurons at the embryonic hindbrain
  per: Ter-Avetisyan, Gohar, et al.
  Publicat: (2013)
• Molecular Analysis of Sensory Axon Branching Unraveled a cGMP-Dependent Signaling Cascade
  per: Dumoulin, Alexandre, et al.
  Publicat: (2018)
• The receptor guanylyl cyclase Npr2 is essential for sensory axon bifurcation within the spinal cord
  per: Schmidt, Hannes, et al.
  Publicat: (2007)
• Regulation of the Natriuretic Peptide Receptor 2 (Npr2) by Phosphorylation of Juxtamembrane Serine and Threonine Residues Is Essential for Bifurcation of Sensory Axons
  per: Schmidt, Hannes, et al.
  Publicat: (2018)