T Cell-Independent Gamma Interferon and B Cells Cooperate To Prevent Mortality Associated with Disseminated Chlamydia muridarum Genital Tract Infection

Samankaltaisia teoksia

• Type I Interferon Signaling Exacerbates Chlamydia muridarum Genital Infection in a Murine Model
  Tekijä: Nagarajan, Uma M., et al.
  Julkaistu: (2008)
• CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection()
  Tekijä: Frazer, Lauren C., et al.
  Julkaistu: (2013)
• Stimulator of IFN Gene Is Critical for Induction of IFN-β during Chlamydia muridarum Infection
  Tekijä: Prantner, Daniel, et al.
  Julkaistu: (2010)
• Interleukin-17 Contributes to Generation of Th1 Immunity and Neutrophil Recruitment during Chlamydia muridarum Genital Tract Infection but Is Not Required for Macrophage Influx or Normal Resolution of Infection
  Tekijä: Scurlock, Amy M., et al.
  Julkaistu: (2011)
• MyD88 Deficiency Leads to Decreased NK Cell Gamma Interferon Production and T Cell Recruitment during Chlamydia muridarum Genital Tract Infection, but a Predominant Th1 Response and Enhanced Monocytic Inflammation Are Associated with Infection Resolution
  Tekijä: Nagarajan, Uma M., et al.
  Julkaistu: (2011)