T Cell-Independent Gamma Interferon and B Cells Cooperate To Prevent Mortality Associated with Disseminated Chlamydia muridarum Genital Tract Infection

Ítems similars

• Type I Interferon Signaling Exacerbates Chlamydia muridarum Genital Infection in a Murine Model
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• CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection()
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• Stimulator of IFN Gene Is Critical for Induction of IFN-β during Chlamydia muridarum Infection
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• Interleukin-17 Contributes to Generation of Th1 Immunity and Neutrophil Recruitment during Chlamydia muridarum Genital Tract Infection but Is Not Required for Macrophage Influx or Normal Resolution of Infection
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• MyD88 Deficiency Leads to Decreased NK Cell Gamma Interferon Production and T Cell Recruitment during Chlamydia muridarum Genital Tract Infection, but a Predominant Th1 Response and Enhanced Monocytic Inflammation Are Associated with Infection Resolution
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