Deficiency of NOD1 Improves the β-Adrenergic Modulation of Ca(2+) Handling in a Mouse Model of Heart Failure

Heart failure (HF) is a complex syndrome characterized by cardiac dysfunction, Ca(2+) mishandling, and chronic activation of the innate immune system. Reduced cardiac output in HF leads to compensatory mechanisms via activation of the adrenergic nervous system. In turn, chronic adrenergic overstimul...

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Detalhes bibliográficos
Publicado no:Front Physiol
Main Authors: Val-Blasco, Almudena, Navarro-García, Jose A., Tamayo, Maria, Piedras, Maria J., Prieto, Patricia, Delgado, Carmen, Ruiz-Hurtado, Gema, Rozas-Romero, Laura, Gil-Fernández, Marta, Zaragoza, Carlos, Boscá, Lisardo, Fernández-Velasco, María
Formato: Artigo
Idioma:Inglês
Publicado em: Frontiers Media S.A. 2018
Assuntos:
Physiology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6010671/
https://ncbi.nlm.nih.gov/pubmed/29962957
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphys.2018.00702
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