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Methamphetamine augments concurrent astrocyte mitochondrial stress, oxidative burden and antioxidant capacity: tipping the balance in HIV-associated neurodegeneration
Methamphetamine (METH) use, with and without human immunodeficiency virus (HIV)-1 comorbidity, exacerbates neurocognitive decline. Oxidative stress is a probable neurotoxic mechanism during HIV-1 central nervous system infection and METH abuse; as viral proteins, antiretroviral therapy and METH have...
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| Vydáno v: | Neurotox Res |
|---|---|
| Hlavní autoři: | , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2017
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6003420/ https://ncbi.nlm.nih.gov/pubmed/28993979 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12640-017-9812-z |
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