The Increased Endogenous Sulfur Dioxide Acts as a Compensatory Mechanism for the Downregulated Endogenous Hydrogen Sulfide Pathway in the Endothelial Cell Inflammation

Endogenous hydrogen sulfide (H(2)S) and sulfur dioxide (SO(2)) are regarded as important regulators to control endothelial cell function and protect endothelial cell against various injuries. In our present study, we aimed to investigate the effect of endogenous H(2)S on the SO(2) generation in the...

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Vydáno v:Front Immunol
Hlavní autoři: Zhang, Da, Wang, Xiuli, Tian, Xiaoyu, Zhang, Lulu, Yang, Guosheng, Tao, Yinghong, Liang, Chen, Li, Kun, Yu, Xiaoqi, Tang, Xinjing, Tang, Chaoshu, Zhou, Jing, Kong, Wei, Du, Junbao, Huang, Yaqian, Jin, Hongfang
Médium: Artigo
Jazyk:Inglês
Vydáno: Frontiers Media S.A. 2018
Témata:
Immunology
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5936987/
https://ncbi.nlm.nih.gov/pubmed/29760703
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fimmu.2018.00882
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