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S100A8/A9 drives neuroinflammatory priming and protects against anxiety-like behavior after sepsis
Sepsis commonly results in both acute and chronic brain dysfunction, which dramatically increase the morbidity associated with this common disease. Chronic brain dysfunction in animal models of sepsis survival is linked to persistent neuroinflammation and expression of multiple cytokines. We have pr...
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| Publicat a: | J Immunol |
|---|---|
| Autors principals: | , , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2018
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5915914/ https://ncbi.nlm.nih.gov/pubmed/29563178 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1700834 |
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