Acidic bile salts induces mucosal barrier dysfunction through let-7a reduction during gastric carcinogenesis after Helicobacter pylori eradication

Gastric cancer (GC) after eradication for Helicobacter pylori (H.pylori) increases, but its carcinogenesis is not elucidated. It is mainly found in acid non-secretion areas (ANA), as mucosal regeneration in acid secretory areas (AA) after eradication changes the acidity and bile toxicity of gastric...

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Pubblicato in:Oncotarget
Autori principali: Takahashi, Yasushi, Uno, Kaname, Iijima, Katsunori, Abe, Yasuhiko, Koike, Tomoyuki, Asano, Naoki, Asanuma, Kiyotaka, Shimosegawa, Tooru
Natura: Artigo
Lingua:Inglês
Pubblicazione: Impact Journals LLC 2018
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Research Paper
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5915058/
https://ncbi.nlm.nih.gov/pubmed/29719591
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.24725
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