Glaucomatous cell derived matrices differentially modulate non-glaucomatous trabecular meshwork cellular behavior

Ocular hypertension is a causal risk-factor to developing glaucoma. This is associated with stiffening of the trabecular meshwork (TM), the primary site of resistance to aqueous-humor-outflow. The mechanisms underlying this stiffening or how pathologic extracellular matrix (ECM) affects cell functio...

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Bibliographic Details
Published in:Acta Biomater
Main Authors: Raghunathan, VijayKrishna, Benoit, Julia, Kasetti, Ramesh, Zode, Gulab, Salemi, Michelle, Phinney, Brett S, Keller, Kate E, Staverosky, Julia A, Murphy, Christopher J, Acott, Ted, Vranka, Janice
Format: Artigo
Language:Inglês
Published: 2018
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Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC5899949/
https://ncbi.nlm.nih.gov/pubmed/29524673
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.actbio.2018.02.037
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