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RGC-32 deficiency protects endothelial cell from inflammation and attenuates atherosclerosis
OBJECTIVE: The objective of this study is to determine the role and underlying mechanisms of response gene to complement 32 (RGC-32) in atherogenesis. APPROACH AND RESULTS: RGC-32 was mainly expressed in endothelial cells (ECs) of atherosclerotic lesions in both apolipoprotein E-deficient (ApoE(−/−)...
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| Veröffentlicht in: | Arterioscler Thromb Vasc Biol |
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| Hauptverfasser: | , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2018
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5864544/ https://ncbi.nlm.nih.gov/pubmed/29449334 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.117.310656 |
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