Mammalian amyloidogenic proteins promote prion nucleation in yeast

Fibrous cross-β aggregates (amyloids) and their transmissible forms (prions) cause diseases in mammals (including humans) and control heritable traits in yeast. Initial nucleation of a yeast prion by transiently overproduced prion-forming protein or its (typically, QN-rich) prion domain is efficient...

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Bibliografiske detaljer
Udgivet i:J Biol Chem
Main Authors: Chandramowlishwaran, Pavithra, Sun, Meng, Casey, Kristin L., Romanyuk, Andrey V., Grizel, Anastasiya V., Sopova, Julia V., Rubel, Aleksandr A., Nussbaum-Krammer, Carmen, Vorberg, Ina M., Chernoff, Yury O.
Format: Artigo
Sprog:Inglês
Udgivet: American Society for Biochemistry and Molecular Biology 2018
Fag:
Molecular Bases of Disease
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5836139/
https://ncbi.nlm.nih.gov/pubmed/29330303
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.809004
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