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Rhinovirus 3C protease suppresses apoptosis and triggers caspase-independent cell death

Apoptosis and programmed necrosis (necroptosis) determine cell fate, and antagonize infection. Execution of these complementary death pathways involves the formation of receptor-interacting protein kinase 1 (RIPK1) containing complexes. RIPK1 binds to adaptor proteins, such as TRIF (Toll-IL-1 recept...

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Foilsithe in:Cell Death Dis
Main Authors: Lötzerich, Mark, Roulin, Pascal S., Boucke, Karin, Witte, Robert, Georgiev, Oleg, Greber, Urs F.
Formáid: Artigo
Teanga:Inglês
Foilsithe: Nature Publishing Group UK 2018
Ábhair:
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5833640/
https://ncbi.nlm.nih.gov/pubmed/29449668
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-018-0306-6
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