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Deleting HDAC3 rescues long-term memory impairments induced by disruption of the neuron-specific chromatin remodeling subunit BAF53b

Multiple epigenetic mechanisms, including histone acetylation and nucleosome remodeling, are known to be involved in long-term memory formation. Enhancing histone acetylation by deleting histone deacetylases, like HDAC3, typically enhances long-term memory formation. In contrast, disrupting nucleoso...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:Learn Mem
Egile Nagusiak: Shu, Guanhua, Kramár, Enikö A., López, Alberto J., Huynh, Grace, Wood, Marcelo A., Kwapis, Janine L.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Cold Spring Harbor Laboratory Press 2018
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC5817283/
https://ncbi.nlm.nih.gov/pubmed/29449454
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/lm.046920.117
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