Prenatal Exposure to Bisphenol A Disrupts Naturally Occurring Bimodal DNA Methylation at Proximal Promoter of fggy, an Obesity-Relevant Gene Encoding a Carbohydrate Kinase, in Gonadal White Adipose Tissues of CD-1 Mice

Exposure of mammalian fetuses to endocrine disruptors can increase the risk of adult-onset diseases. We previously showed that exposure of mouse fetuses to bisphenol A (BPA) caused adult-onset obesity. To examine roles of epigenetic changes in this delayed toxicity, we determined the effects of feta...

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Detalhes bibliográficos
Publicado no:Endocrinology
Main Authors: Taylor, Julia A., Shioda, Keiko, Mitsunaga, Shino, Yawata, Shiomi, Angle, Brittany M., Nagel, Susan C., vom Saal, Frederick S., Shioda, Toshi
Formato: Artigo
Idioma:Inglês
Publicado em: Endocrine Society 2017
Assuntos:
Research Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5774244/
https://ncbi.nlm.nih.gov/pubmed/29220483
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/en.2017-00711
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