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Mutations in Caenorhabditis elegans neuroligin-like glit-1, the apoptosis pathway and the calcium chaperone crt-1 increase dopaminergic neurodegeneration after 6-OHDA treatment

The loss of dopaminergic neurons is a hallmark of Parkinson’s disease, the aetiology of which is associated with increased levels of oxidative stress. We used C. elegans to screen for genes that protect dopaminergic neurons against oxidative stress and isolated glit-1 (gliotactin (Drosophila neuroli...

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Библиографические подробности
Опубликовано в: :PLoS Genet
Главные авторы: Offenburger, Sarah-Lena, Jongsma, Elisabeth, Gartner, Anton
Формат: Artigo
Язык:Inglês
Опубликовано: Public Library of Science 2018
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC5773152/
https://ncbi.nlm.nih.gov/pubmed/29346364
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pgen.1007106
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