Vitamin D Receptor Activation Influences NADPH Oxidase (NOX(2)) Activity and Protects against Neurological Deficits and Apoptosis in a Rat Model of Traumatic Brain Injury

Traumatic brain injury (TBI) is a worldwide phenomenon which results in significant neurological and cognitive deficits in humans. Vitamin D (VD) is implicated as a therapeutic strategy for various neurological diseases now. Recently, inhibition of the NADPH oxidase (NOX(2)) was reported to protect...

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Dettagli Bibliografici
Pubblicato in:Oxid Med Cell Longev
Autori principali: Cui, Changmeng, Song, Sixin, Cui, Jianzhong, Feng, Yan, Gao, Junling, Jiang, Pei
Natura: Artigo
Lingua:Inglês
Pubblicazione: Hindawi 2017
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5749321/
https://ncbi.nlm.nih.gov/pubmed/29410737
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2017/9245702
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