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Inhalation of fine particulate matter impairs endothelial progenitor cell function via pulmonary oxidative stress

OBJECTIVE: Exposure to fine particulate matter (PM(2.5)) air pollution is associated with the depletion of circulating endothelial progenitor cells (EPCs) as well as vascular injury and dysfunction. Nevertheless, it remains unclear whether PM(2.5) exposure leads to significant impairments in EPC fun...

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Dades bibliogràfiques
Publicat a:Arterioscler Thromb Vasc Biol
Autors principals: Haberzettl, Petra, Conklin, Daniel J., Abplanalp, Wesley T., Bhatnagar, Aruni, O’Toole, Timothy E.
Format: Artigo
Idioma:Inglês
Publicat: 2017
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5746456/
https://ncbi.nlm.nih.gov/pubmed/29191925
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.117.309971
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