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Inhalation of fine particulate matter impairs endothelial progenitor cell function via pulmonary oxidative stress
OBJECTIVE: Exposure to fine particulate matter (PM(2.5)) air pollution is associated with the depletion of circulating endothelial progenitor cells (EPCs) as well as vascular injury and dysfunction. Nevertheless, it remains unclear whether PM(2.5) exposure leads to significant impairments in EPC fun...
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| Publicat a: | Arterioscler Thromb Vasc Biol |
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| Autors principals: | , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2017
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5746456/ https://ncbi.nlm.nih.gov/pubmed/29191925 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.117.309971 |
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