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Inhalation of fine particulate matter impairs endothelial progenitor cell function via pulmonary oxidative stress

OBJECTIVE: Exposure to fine particulate matter (PM(2.5)) air pollution is associated with the depletion of circulating endothelial progenitor cells (EPCs) as well as vascular injury and dysfunction. Nevertheless, it remains unclear whether PM(2.5) exposure leads to significant impairments in EPC fun...

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Bibliografski detalji
Izdano u:Arterioscler Thromb Vasc Biol
Glavni autori: Haberzettl, Petra, Conklin, Daniel J., Abplanalp, Wesley T., Bhatnagar, Aruni, O’Toole, Timothy E.
Format: Artigo
Jezik:Inglês
Izdano: 2017
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5746456/
https://ncbi.nlm.nih.gov/pubmed/29191925
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.117.309971
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