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Inhalation of fine particulate matter impairs endothelial progenitor cell function via pulmonary oxidative stress
OBJECTIVE: Exposure to fine particulate matter (PM(2.5)) air pollution is associated with the depletion of circulating endothelial progenitor cells (EPCs) as well as vascular injury and dysfunction. Nevertheless, it remains unclear whether PM(2.5) exposure leads to significant impairments in EPC fun...
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| Publicado no: | Arterioscler Thromb Vasc Biol |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5746456/ https://ncbi.nlm.nih.gov/pubmed/29191925 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.117.309971 |
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