2, 3, 7, 8‐Tetrachlorodibenzo‐p‐dioxin promotes endothelial cell apoptosis through activation of EP3/p38MAPK/Bcl‐2 pathway

Endothelial injury or dysfunction is an early event in the pathogenesis of atherosclerosis. Epidemiological and animal studies have shown that 2, 3, 7, 8‐tetrachlorodibenzo‐p‐dioxin (TCDD) exposure increases morbidity and mortality from chronic cardiovascular diseases, including atherosclerosis. How...

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Publicado en:J Cell Mol Med
Autores principales: Yu, Yu, Liu, Qian, Guo, Shumin, Zhang, Qianqian, Tang, Juan, Liu, Guizhu, Kong, Deping, Li, Juanjuan, Yan, Shuai, Wang, Ruiguo, Wang, Peilong, Su, Xiaoou, Yu, Ying
Formato: Artigo
Lenguaje:Inglês
Publicado: John Wiley and Sons Inc. 2017
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Original Articles
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC5706494/
https://ncbi.nlm.nih.gov/pubmed/28699682
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.13265
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