G2A Attenuates Propionibacterium acnes Induction of Inflammatory Cytokines in Human Monocytes

BACKGROUND: Acne vulgaris is a disease of the pilosebaceous unit characterized by increased sebum production, hyperkeratinization, and immune responses to Propionibacterium acnes (PA). Here, we explore a possible mechanism by which a lipid receptor, G2A, regulates immune responses to a commensal bac...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Ann Dermatol
Päätekijät: Park, Andrew J, Agak, George W, Qin, Min, Hisaw, Lisa D, Pirouz, Aslan, Kao, Stephanie, Marinelli, Laura J, Garbán, Hermes J, Thiboutot, Diane, Liu, Philip T, Kim, Jenny
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: The Korean Dermatological Association; The Korean Society for Investigative Dermatology 2017
Aiheet:
Original Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5705349/
https://ncbi.nlm.nih.gov/pubmed/29200756
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.5021/ad.2017.29.6.688
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