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Bcl6 gene-silencing facilitates PMA-induced megakaryocyte differentiation in K562 cells
Targeted therapy via imatinib appears to be a promising approach for chronic myeloid leukemia (CML) therapy. However, refractory and resistance to imatinib therapy has encouraged many investigators to get involved in development of new therapeutic agents such as Phorbol 12-myrestrat 13-acetate (PMA)...
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| Опубликовано в: : | J Cell Commun Signal |
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| Главные авторы: | , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
Springer Netherlands
2017
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5704040/ https://ncbi.nlm.nih.gov/pubmed/28589317 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12079-017-0395-5 |
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