Endothelial α6β4 integrin protects during experimental autoimmune encephalomyelitis-induced neuroinflammation by maintaining vascular integrity and tight junction protein expression

Lignende værker

• Absence of endothelial α5β1 integrin triggers early onset of experimental autoimmune encephalomyelitis due to reduced vascular remodeling and compromised vascular integrity
  af: Kant, Ravi, et al.
  Udgivet: (2019)
• Extensive vascular remodeling in the spinal cord of pre-symptomatic experimental autoimmune encephalomyelitis mice; increased vessel expression of fibronectin and the α5β1 integrin
  af: Boroujerdi, Amin, et al.
  Udgivet: (2013)
• Hypoxic pre-conditioning suppresses experimental autoimmune encephalomyelitis by modifying multiple properties of blood vessels
  af: Halder, Sebok K., et al.
  Udgivet: (2018)
• Activated Protein C Attenuates Experimental Autoimmune Encephalomyelitis Progression by Enhancing Vascular Integrity and Suppressing Microglial Activation
  af: Kant, Ravi, et al.
  Udgivet: (2020)
• Chronic mild hypoxia promotes profound vascular remodeling in spinal cord blood vessels, preferentially in white matter, via an α5β1 integrin-mediated mechanism
  af: Halder, Sebok K., et al.
  Udgivet: (2018)