Methylglyoxal‐induced apoptosis is dependent on the suppression of c‐FLIP(L) expression via down‐regulation of p65 in endothelial cells

Methylglyoxal (MGO) is a reactive dicarbonyl metabolite of glucose, and its plasma levels are elevated in patients with diabetes. Studies have shown that MGO combines with the amino and sulphhydryl groups of proteins to form stable advanced glycation end products (AGEs), which are associated with va...

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Veröffentlicht in:J Cell Mol Med
Hauptverfasser: Jang, Ji Hoon, Kim, Eun‐Ae, Park, Hye‐Jin, Sung, Eon‐Gi, Song, In‐Hwan, Kim, Joo‐Young, Woo, Chang‐Hoon, Doh, Kyung‐Oh, Kim, Kook Hyun, Lee, Tae‐Jin
Format: Artigo
Sprache:Inglês
Veröffentlicht: John Wiley and Sons Inc. 2017
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Original Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5661116/
https://ncbi.nlm.nih.gov/pubmed/28444875
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.13188
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