Methylglyoxal‐induced apoptosis is dependent on the suppression of c‐FLIP(L) expression via down‐regulation of p65 in endothelial cells

Methylglyoxal (MGO) is a reactive dicarbonyl metabolite of glucose, and its plasma levels are elevated in patients with diabetes. Studies have shown that MGO combines with the amino and sulphhydryl groups of proteins to form stable advanced glycation end products (AGEs), which are associated with va...

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Detaylı Bibliyografya
Yayımlandı:J Cell Mol Med
Asıl Yazarlar: Jang, Ji Hoon, Kim, Eun‐Ae, Park, Hye‐Jin, Sung, Eon‐Gi, Song, In‐Hwan, Kim, Joo‐Young, Woo, Chang‐Hoon, Doh, Kyung‐Oh, Kim, Kook Hyun, Lee, Tae‐Jin
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: John Wiley and Sons Inc. 2017
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5661116/
https://ncbi.nlm.nih.gov/pubmed/28444875
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.13188
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