Activation of CaMKIIγ potentiates T-cell acute lymphoblastic leukemia leukemogenesis via phosphorylating FOXO3a

Ca(2+)/calmodulin–dependent protein kinase II γ (CaMKIIγ) can regulate the proliferation and differentiation of myeloid leukemia cells and accelerate chronic myeloid leukemia blast crisis, but the role of CaMKIIγ in T-cell acute lymphoblastic leukemia (T-ALL) leukemogenesis remains poorly understood...

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Foilsithe in:Oncotarget
Main Authors: Jiang, Xudong, Wu, Zhaoxing, Lu, Xiaoya, Zhang, Xuzhao, Yu, Qingfeng, Gan, Yichao, Wu, Bowen, Xu, Ying, Zheng, Weiwei, Zhang, Lei, Xu, Fei, Ma, An, Gan, Xiaoxian, Huang, Silvia, Yu, Xiaofang, Huang, Wendong, Xu, Rongzhen
Formáid: Artigo
Teanga:Inglês
Foilsithe: Impact Journals LLC 2017
Ábhair:
Research Paper
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5650399/
https://ncbi.nlm.nih.gov/pubmed/29088844
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.20504
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