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Mice lacking GRIP1/2 show increased social interactions and enhanced phosphorylation at GluA2-S880
Glutamate receptor interacting proteins 1 and 2 (GRIP1/2) play an important role in regulating synaptic trafficking of AMPA receptor 2/3 (GluA2/3) and synaptic strength. Gain-of-function GRIP1 mutations are implicated in social behavioral deficits in autism. To study mechanisms of Grip1/2-mediated A...
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| 發表在: | Behav Brain Res |
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| Main Authors: | , , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
2017
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5628023/ https://ncbi.nlm.nih.gov/pubmed/28063882 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbr.2016.12.042 |
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