H2Av facilitates H3S10 phosphorylation but is not required for heat shock-induced chromatin decondensation or transcriptional elongation

A model has been proposed in which JIL-1 kinase-mediated H3S10 and H2Av phosphorylation is required for transcriptional elongation and heat shock-induced chromatin decondensation. However, here we show that although H3S10 phosphorylation is indeed compromised in the H2Av null mutant, chromatin decon...

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Detalhes bibliográficos
Publicado no:Development
Main Authors: Li, Yeran, Wang, Chao, Cai, Weili, Sengupta, Saheli, Zavortink, Michael, Deng, Huai, Girton, Jack, Johansen, Jørgen, Johansen, Kristen M.
Formato: Artigo
Idioma:Inglês
Publicado em: The Company of Biologists Ltd 2017
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5612252/
https://ncbi.nlm.nih.gov/pubmed/28807902
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/dev.151134
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