Restraint of presynaptic protein levels by Wnd/DLK signaling mediates synaptic defects associated with the kinesin-3 motor Unc-104

The kinesin-3 family member Unc-104/KIF1A is required for axonal transport of many presynaptic components to synapses, and mutation of this gene results in synaptic dysfunction in mice, flies and worms. Our studies at the Drosophila neuromuscular junction indicate that many synaptic defects in unc-1...

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Detalhes bibliográficos
Publicado no:eLife
Main Authors: Li, Jiaxing, Zhang, Yao V, Asghari Adib, Elham, Stanchev, Doychin T, Xiong, Xin, Klinedinst, Susan, Soppina, Pushpanjali, Jahn, Thomas Robert, Hume, Richard I, Rasse, Tobias M, Collins, Catherine A
Formato: Artigo
Idioma:Inglês
Publicado em: eLife Sciences Publications, Ltd 2017
Assuntos:
Cell Biology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5605197/
https://ncbi.nlm.nih.gov/pubmed/28925357
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.24271
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