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Autocrine activation of the IFN signaling pathway may promote immune escape in glioblastoma
BACKGROUND: Interferons (IFNs) are cytokines typically induced upon viral infection but are constitutively expressed also in the absence of acute infection. The physiological role of autocrine and paracrine IFN signaling, however, remains poorly understood, and its function in glioblastoma has not b...
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| Pubblicato in: | Neuro Oncol |
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| Autori principali: | , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Oxford University Press
2017
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5596176/ https://ncbi.nlm.nih.gov/pubmed/28475775 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/nox051 |
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