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The spread of prion-like proteins by lysosomes and tunneling nanotubes: Implications for neurodegenerative diseases
Progression of pathology in neurodegenerative diseases is hypothesized to be a non–cell-autonomous process that may be mediated by the productive spreading of prion-like protein aggregates from a “donor cell” that is the source of misfolded aggregates to an “acceptor cell” in which misfolding is pro...
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| Pubblicato in: | J Cell Biol |
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| Autori principali: | , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
The Rockefeller University Press
2017
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5584166/ https://ncbi.nlm.nih.gov/pubmed/28724527 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.201701047 |
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