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p16(Ink4a)-induced senescence of pancreatic beta cells enhances insulin secretion
Cellular senescence is thought to contribute to age-associated deterioration of tissue physiology. The senescence effector p16(Ink4a) is expressed in pancreatic beta cells during aging and limits their proliferative potential; however, its effects on beta cell function are poorly characterized. We f...
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| Udgivet i: | Nat Med |
|---|---|
| Main Authors: | , , , , , , , , , , , , , , , , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2016
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5546206/ https://ncbi.nlm.nih.gov/pubmed/26950362 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm.4054 |
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