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Kininase 1 As a Preclinical Therapeutic Target for Kinin B(1) Receptor in Insulin Resistance
Kinin B1 receptor (B1R) contributes to insulin resistance, an early event in type 2 diabetes, through the upregulation and activation of the inducible form of nitric oxide synthase (iNOS), pro-inflammatory cytokines and the oxidative stress. This study addresses the hypothesis that inhibition of kin...
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| 出版年: | Front Pharmacol |
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| 主要な著者: | , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Frontiers Media S.A.
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5539221/ https://ncbi.nlm.nih.gov/pubmed/28824433 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphar.2017.00509 |
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