Glucose-induced translocation of protein kinase C in rat pancreatic islets.
The role of protein kinase C (PKC) as a mediator of glucose-induced insulin secretion has been a subject of controversy. Glucose-induced translocation of PKC has not been reported, and the relevant PKC isoenzymes in islets have not been identified. To address these issues, we developed specific anti...
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| Veröffentlicht in: | Proc Natl Acad Sci U S A |
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| Hauptverfasser: | , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
National Academy of Sciences
1990
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| Schlagworte: | |
| Online-Zugang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC55280/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2263640/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.87.24.9893 |
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