Oncogenic STAT5 signaling promotes oxidative stress in chronic myeloid leukemia cells by repressing antioxidant defenses

STAT5 transcription factors are frequently activated in hematopoietic neoplasms and are targets of various tyrosine kinase oncogenes. Evidences for a crosstalk between STAT5 and reactive oxygen species (ROS) metabolism have recently emerged but mechanisms involved in STAT5-mediated regulation of ROS...

Descripció completa

Guardat en:
Dades bibliogràfiques
Publicat a:Oncotarget
Autors principals: Bourgeais, Jerome, Ishac, Nicole, Medrzycki, Magdalena, Brachet-Botineau, Marie, Desbourdes, Laura, Gouilleux-Gruart, Valerie, Pecnard, Emmanuel, Rouleux-Bonnin, Florence, Gyan, Emmanuel, Domenech, Jorge, Mazurier, Frederic, Moriggl, Richard, Bunting, Kevin D., Herault, Olivier, Gouilleux, Fabrice
Format: Artigo
Idioma:Inglês
Publicat: Impact Journals LLC 2016
Matèries:
Research Paper
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5522035/
https://ncbi.nlm.nih.gov/pubmed/27566554
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.11480
Etiquetes: Afegir etiqueta
Sense etiquetes, Sigues el primer a etiquetar aquest registre!

Ítems similars