טוען...
Administration of tauroursodeoxycholic acid prevents endothelial dysfunction caused by an oral glucose load
Postprandial hyperglycemia leads to a transient impairment in endothelial function; however, the mechanisms remain largely unknown. Previous work in cell culture models demonstrate that high glucose results in endoplasmic reticulum (ER) stress and, in animal studies, ER stress has been implicated as...
שמור ב:
| הוצא לאור ב: | Clin Sci (Lond) |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2016
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5507333/ https://ncbi.nlm.nih.gov/pubmed/27503949 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/CS20160501 |
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