Neuronal Damage Induced by Perinatal Asphyxia Is Attenuated by Postinjury Glutaredoxin-2 Administration

The general disruption of redox signaling following an ischemia-reperfusion episode has been proposed as a crucial component in neuronal death and consequently brain damage. Thioredoxin (Trx) family proteins control redox reactions and ensure protein regulation via specific, oxidative posttranslatio...

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Dettagli Bibliografici
Pubblicato in:Oxid Med Cell Longev
Autori principali: Romero, Juan Ignacio, Holubiec, Mariana Inés, Tornatore, Tamara Logica, Rivière, Stéphanie, Hanschmann, Eva-Maria, Kölliker-Frers, Rodolfo Alberto, Tau, Julia, Blanco, Eduardo, Galeano, Pablo, Rodríguez de Fonseca, Fernando, Lillig, Christopher Horst, Capani, Francisco
Natura: Artigo
Lingua:Inglês
Pubblicazione: Hindawi 2017
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5494587/
https://ncbi.nlm.nih.gov/pubmed/28706574
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2017/4162465
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