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Glucose sensing by skeletal myocytes couples nutrient signaling to systemic homeostasis
Skeletal muscle is a major site of postprandial glucose disposal. Inadequate insulin action in skeletal myocytes contributes to hyperglycemia in diabetes. While glucose is known to stimulate insulin secretion by β cells, whether it directly engages nutrient signaling pathways in skeletal muscle to m...
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| 出版年: | Mol Cell |
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| 主要な著者: | , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5489118/ https://ncbi.nlm.nih.gov/pubmed/28475869 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2017.04.007 |
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