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Upregulation and phosphorylation of HspB1/Hsp25 and HspB5/αB-crystallin after transient middle cerebral artery occlusion in rats

Ischemic stroke leads to cellular dysfunction, cell death, and devastating clinical outcomes. The cells of the brain react to such a cellular stress by a stress response with an upregulation of heat shock proteins resulting in activation of endogenous neuroprotective capacities. Several members of t...

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Detalhes bibliográficos
Publicado no:Cell Stress Chaperones
Main Authors: Bartelt-Kirbach, Britta, Slowik, Alexander, Beyer, Cordian, Golenhofen, Nikola
Formato: Artigo
Idioma:Inglês
Publicado em: Springer Netherlands 2017
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5465040/
https://ncbi.nlm.nih.gov/pubmed/28425051
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12192-017-0794-9
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