TSA protects H9c2 cells against thapsigargin-induced apoptosis related to endoplasmic reticulum stress-mediated mitochondrial injury

Endoplasmic reticulum stress (ERS) activates an adaptive unfolded protein response (UPR) that facilitates cellular repair, however, under prolonged ER stress, the UPR can ultimately trigger apoptosis thereby terminating damaged cells. Recently, TSA has shown protective effects on ERS and its mechani...

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Publicat a:Saudi Pharm J
Autors principals: Li, Zhiping, Liu, Yan, Dai, Xinlun, Zhou, Qiangqiang, Liu, Xueli, Li, Zeyu, Chen, Xia
Format: Artigo
Idioma:Inglês
Publicat: Elsevier 2017
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5447435/
https://ncbi.nlm.nih.gov/pubmed/28579897
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jsps.2017.04.029
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