Φορτώνει......
Gap junction remodelling in human heart failure is associated with increased interaction of connexin43 with ZO-1
AIMS: Remodelling of gap junctions, involving reduction of total gap junction quantity and down-regulation of connexin43 (Cx43), contributes to the arrhythmic substrate in congestive heart failure. However, little is known of the underlying mechanisms. Recent studies from in vitro systems suggest th...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Cardiovasc Res |
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| Κύριοι συγγραφείς: | , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Oxford University Press
2008
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5436744/ https://ncbi.nlm.nih.gov/pubmed/18056766 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvm083 |
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