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Regulation of CHK1 by mTOR contributes to the evasion of DNA damage barrier of cancer cells

Oncogenic transformation leads to dysregulated cell proliferation, nutrient deficiency, and hypoxia resulting in metabolic stress and increased DNA damage. In normal cells, such metabolic stress leads to inhibition of signaling through the mammalian Target of Rapamycin Complex 1 (mTORC1), reduction...

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Dettagli Bibliografici
Pubblicato in:Sci Rep
Autori principali: Zhou, Xinhui, Liu, Weijin, Hu, Xing, Dorrance, Adrienne, Garzon, Ramiro, Houghton, Peter J., Shen, Changxian
Natura: Artigo
Lingua:Inglês
Pubblicazione: Nature Publishing Group UK 2017
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5431544/
https://ncbi.nlm.nih.gov/pubmed/28484242
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-017-01729-w
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