Acute cigarette smoke exposure activates apoptotic and inflammatory programs but a second stimulus is required to induce epithelial to mesenchymal transition in COPD epithelium

BACKGROUND: Smoking and aberrant epithelial responses are risk factors for lung cancer as well as chronic obstructive pulmonary disease and idiopathic pulmonary fibrosis. In these conditions, disease progression is associated with epithelial damage and fragility, airway remodelling and sub-epithelia...

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Detalhes bibliográficos
Publicado no:Respir Res
Main Authors: Murray, Lynne A., Dunmore, Rebecca, Camelo, Ana, Da Silva, Carla A., Gustavsson, Malin J., Habiel, David M., Hackett, Tillie L, Hogaboam, Cory M., Sleeman, Matthew A., Knight, Darryl A.
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2017
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5415733/
https://ncbi.nlm.nih.gov/pubmed/28468623
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12931-017-0565-2
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