Acute cigarette smoke exposure activates apoptotic and inflammatory programs but a second stimulus is required to induce epithelial to mesenchymal transition in COPD epithelium

BACKGROUND: Smoking and aberrant epithelial responses are risk factors for lung cancer as well as chronic obstructive pulmonary disease and idiopathic pulmonary fibrosis. In these conditions, disease progression is associated with epithelial damage and fragility, airway remodelling and sub-epithelia...

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Библиографические подробности
Опубликовано в: :Respir Res
Главные авторы: Murray, Lynne A., Dunmore, Rebecca, Camelo, Ana, Da Silva, Carla A., Gustavsson, Malin J., Habiel, David M., Hackett, Tillie L, Hogaboam, Cory M., Sleeman, Matthew A., Knight, Darryl A.
Формат: Artigo
Язык:Inglês
Опубликовано: BioMed Central 2017
Предметы:
Research
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC5415733/
https://ncbi.nlm.nih.gov/pubmed/28468623
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12931-017-0565-2
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