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Expanded polyalanine tracts function as nuclear export signals and promote protein mislocalization via eEF1A1 factor
Polyalanine (poly(A)) diseases are caused by the expansion of translated GCN triplet nucleotide sequences encoding poly(A) tracts in proteins. To date, nine human disorders have been found to be associated with poly(A) tract expansions, including congenital central hypoventilation syndrome and oculo...
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| 出版年: | J Biol Chem |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Biochemistry and Molecular Biology
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5392573/ https://ncbi.nlm.nih.gov/pubmed/28246169 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M116.763599 |
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