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BVES regulates c-Myc stability via PP2A and suppresses colitis-induced tumorigenesis
OBJECTIVE: Blood vessel epicardial substance (BVES) is a tight junction-associated protein that regulates epithelial-mesenchymal states and is underexpressed in epithelial malignancy. However, the functional impact of BVES loss on tumorigenesis is unknown. Here we define the in vivo role of BVES in...
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| I publikationen: | Gut |
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| Huvudupphovsmän: | , , , , , , , , , , , , , , , , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2016
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5385850/ https://ncbi.nlm.nih.gov/pubmed/28389570 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1136/gutjnl-2015-310255 |
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