BVES regulates c-Myc stability via PP2A and suppresses colitis-induced tumorigenesis

OBJECTIVE: Blood vessel epicardial substance (BVES) is a tight junction-associated protein that regulates epithelial-mesenchymal states and is underexpressed in epithelial malignancy. However, the functional impact of BVES loss on tumorigenesis is unknown. Here we define the in vivo role of BVES in...

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Bibliografiska uppgifter
I publikationen:Gut
Huvudupphovsmän: Parang, Bobak, Kaz, Andrew M., Barrett, Caitlyn W., Short, Sarah P., Ning, Wei, Keating, Cody E., Mittal, Mukul K., Naik, Rishi D., Washington, Mary K., Revetta, Frank L., Smith, J. Joshua, Chen, Xi, Wilson, Keith T., Brand, Thomas, Bader, David M., Tansey, William P., Chen, Ru, Brentnall, Teresa A., Grady, William M., Williams, Christopher S.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2016
Ämnen:
Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC5385850/
https://ncbi.nlm.nih.gov/pubmed/28389570
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1136/gutjnl-2015-310255
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