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HIV-Tat protein and amyloid β peptide form multifibrillar structures that cause neurotoxicity
We investigated direct interactions between the human immunodeficiency virus (HIV)-trans-activator of transcription (Tat) protein and amyloid β peptide. Amyloid β-Tat complexes are readily formed extracellularly in the brain. In vitro studies showed that in the presence of Tat, the uniform amyloid f...
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| Publicado no: | Nat Struct Mol Biol |
|---|---|
| Main Authors: | , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5383535/ https://ncbi.nlm.nih.gov/pubmed/28218748 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nsmb.3379 |
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