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KChIP2 regulates the cardiac Ca(2+) transient and myocyte contractility by targeting ryanodine receptor activity
Pathologic electrical remodeling and attenuated cardiac contractility are featured characteristics of heart failure. Coinciding with these remodeling events is a loss of the K(+) channel interacting protein, KChIP2. While, KChIP2 enhances the expression and stability of the Kv4 family of potassium c...
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| Vydáno v: | PLoS One |
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| Hlavní autoři: | , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Public Library of Science
2017
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5383259/ https://ncbi.nlm.nih.gov/pubmed/28384221 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0175221 |
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