Intact mitochondrial Ca(2+) uniport is essential for agonist-induced activation of endothelial nitric oxide synthase (eNOS)

Mitochondrial Ca(2+) uptake regulates diverse endothelial cell functions and has also been related to nitric oxide (NO(•)) production. However, it is not entirely clear if the organelles support or counteract NO(•) biosynthesis by taking up Ca(2+). The objective of this study was to verify whether o...

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Bibliografiska uppgifter
I publikationen:Free Radic Biol Med
Huvudupphovsmän: Charoensin, Suphachai, Eroglu, Emrah, Opelt, Marissa, Bischof, Helmut, Madreiter-Sokolowski, Corina T., Kirsch, Andrijana, Depaoli, Maria R., Frank, Saša, Schrammel, Astrid, Mayer, Bernd, Waldeck-Weiermair, Markus, Graier, Wolfgang F., Malli, Roland
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2016
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC5381715/
https://ncbi.nlm.nih.gov/pubmed/27923677
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2016.11.049
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