Intact mitochondrial Ca(2+) uniport is essential for agonist-induced activation of endothelial nitric oxide synthase (eNOS)

Mitochondrial Ca(2+) uptake regulates diverse endothelial cell functions and has also been related to nitric oxide (NO(•)) production. However, it is not entirely clear if the organelles support or counteract NO(•) biosynthesis by taking up Ca(2+). The objective of this study was to verify whether o...

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Gepubliceerd in:Free Radic Biol Med
Hoofdauteurs: Charoensin, Suphachai, Eroglu, Emrah, Opelt, Marissa, Bischof, Helmut, Madreiter-Sokolowski, Corina T., Kirsch, Andrijana, Depaoli, Maria R., Frank, Saša, Schrammel, Astrid, Mayer, Bernd, Waldeck-Weiermair, Markus, Graier, Wolfgang F., Malli, Roland
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2016
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Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5381715/
https://ncbi.nlm.nih.gov/pubmed/27923677
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2016.11.049
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