Intact mitochondrial Ca(2+) uniport is essential for agonist-induced activation of endothelial nitric oxide synthase (eNOS)

Mitochondrial Ca(2+) uptake regulates diverse endothelial cell functions and has also been related to nitric oxide (NO(•)) production. However, it is not entirely clear if the organelles support or counteract NO(•) biosynthesis by taking up Ca(2+). The objective of this study was to verify whether o...

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Shranjeno v:
Bibliografske podrobnosti
izdano v:Free Radic Biol Med
Main Authors: Charoensin, Suphachai, Eroglu, Emrah, Opelt, Marissa, Bischof, Helmut, Madreiter-Sokolowski, Corina T., Kirsch, Andrijana, Depaoli, Maria R., Frank, Saša, Schrammel, Astrid, Mayer, Bernd, Waldeck-Weiermair, Markus, Graier, Wolfgang F., Malli, Roland
Format: Artigo
Jezik:Inglês
Izdano: 2016
Teme:
Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC5381715/
https://ncbi.nlm.nih.gov/pubmed/27923677
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2016.11.049
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