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BCA2/Rabring7 Interferes with HIV-1 Proviral Transcription by Enhancing the SUMOylation of IκBα

BCA2/Rabring7 is a BST2 cofactor that promotes the lysosomal degradation of trapped HIV-1 virions but also functions as a BST2-independent anti-HIV factor by targeting Gag for lysosomal degradation. Since many antiviral factors regulate the NF-κB innate signaling pathway, we investigated whether BCA...

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Detalhes bibliográficos
Publicado no:J Virol
Main Authors: Colomer-Lluch, Marta, Serra-Moreno, Ruth
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology 2017
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5375697/
https://ncbi.nlm.nih.gov/pubmed/28122985
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.02098-16
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