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Post-traumatic cytotoxic edema is directly related to mitochondrial function
Cerebral edema represents a major threat following traumatic brain injury. However, therapeutic measures for control of intracranial pressure alone have failed to restore cerebral metabolism and improve neurological outcome. Since mitochondrial damage results in ATP depletion and deactivation of mem...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | J Cereb Blood Flow Metab |
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| Κύριοι συγγραφείς: | , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
SAGE Publications
2015
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5363733/ https://ncbi.nlm.nih.gov/pubmed/26672111 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X15621068 |
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