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Chronic Ca(2+) influx through voltage-dependent Ca(2+) channels enhance delayed rectifier K(+) currents via activating Src family tyrosine kinase in rat hippocampal neurons
Excessive influx and the subsequent rapid cytosolic elevation of Ca(2+) in neurons is the major cause to induce hyperexcitability and irreversible cell damage although it is an essential ion for cellular signalings. Therefore, most neurons exhibit several cellular mechanisms to homeostatically regul...
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| Udgivet i: | Korean J Physiol Pharmacol |
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| Main Authors: | , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
The Korean Physiological Society and The Korean Society of Pharmacology
2017
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5343060/ https://ncbi.nlm.nih.gov/pubmed/28280420 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4196/kjpp.2017.21.2.259 |
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