Chronic Ca(2+) influx through voltage-dependent Ca(2+) channels enhance delayed rectifier K(+) currents via activating Src family tyrosine kinase in rat hippocampal neurons

Excessive influx and the subsequent rapid cytosolic elevation of Ca(2+) in neurons is the major cause to induce hyperexcitability and irreversible cell damage although it is an essential ion for cellular signalings. Therefore, most neurons exhibit several cellular mechanisms to homeostatically regul...

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Detalhes bibliográficos
Publicado no:Korean J Physiol Pharmacol
Principais autores: Yang, Yoon-Sil, Jeon, Sang-Chan, Kim, Dong-Kwan, Eun, Su-Yong, Jung, Sung-Cherl
Formato: Artigo
Idioma:Inglês
Publicado em: The Korean Physiological Society and The Korean Society of Pharmacology 2017
Assuntos:
Original Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5343060/
https://ncbi.nlm.nih.gov/pubmed/28280420
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4196/kjpp.2017.21.2.259
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